Synopsis

Source of validity: Scientific
Rating (out of 5): 5

Vitamin B2, also known as riboflavin, is scientifically validated as essential for mitochondrial function. Riboflavin is a precursor for the cofactors flavin mononucleotide (FMN) and flavin adenine dinucleotide (FAD), both of which are critical for the activity of various enzymes in the mitochondrial electron transport chain. These enzymes are involved in oxidative phosphorylation, the process through which mitochondria generate ATP, the primary energy currency of the cell. Deficiency of riboflavin impairs mitochondrial energy production, leading to symptoms such as fatigue, muscle weakness, and in severe cases, neurological disorders. Clinical studies and biochemical research have demonstrated that riboflavin supplementation can help improve mitochondrial diseases associated with complex I and complex II deficiencies and is sometimes part of therapeutic regimens for conditions such as mitochondrial myopathies and multiple acyl-CoA dehydrogenase deficiency (MADD). The scientific evidence supporting the role of vitamin B2 in supporting mitochondrial health is robust, rooted in well-understood biochemistry and clinical applications, making its use in this context highly justified.

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