Synopsis

Source of validity: Scientific
Rating (out of 5): 3

Vitamin K, particularly in its K2 (menaquinone) form, has garnered scientific interest for its role in bone health. Vitamin K is essential for the carboxylation of osteocalcin, a protein involved in bone mineralization. This process allows osteocalcin to bind calcium effectively, which is crucial for bone strength and repair. Several observational studies have linked higher dietary intake or serum levels of vitamin K to improved bone mineral density and a lower risk of fractures, particularly in postmenopausal women. Randomized controlled trials (RCTs) have shown mixed results: some indicate that vitamin K supplementation (especially K2) can reduce fracture risk and improve bone density, while others show little or no effect, particularly when adequate vitamin D and calcium are present.

Meta-analyses suggest potential benefits of vitamin K (mainly K2) supplementation for bone health and fracture reduction, but the quality of evidence is moderate, and professional guidelines do not universally recommend vitamin K supplementation for bone fracture treatment or prevention outside of deficiency states. Nonetheless, there is a plausible biological mechanism and a growing body of research supporting its adjunctive use in bone health and healing. Thus, while not a standard treatment, vitamin K's use in supporting broken bone healing is scientifically plausible and supported by moderate evidence, meriting further investigation.

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