Synopsis

Source of validity: Scientific
Rating (out of 5): 2

L-glutathione is an antioxidant naturally produced in the body, and its primary role is to neutralize free radicals and reduce oxidative stress. There is some scientific interest in using L-glutathione to support autoimmune disorders, as oxidative stress and inflammation are known contributors to the pathogenesis of many autoimmune conditions such as rheumatoid arthritis, systemic lupus erythematosus, and multiple sclerosis. A few small studies and preclinical models have suggested that glutathione levels tend to be lower in individuals with certain autoimmune diseases, and that restoring glutathione may help reduce disease activity by modulating immune responses and blocking inflammatory cytokines. However, most of this evidence comes from either animal studies, cell culture experiments, or small pilot studies in humans. Large, well-controlled clinical trials demonstrating clear benefit in autoimmune patients are lacking. Thus, while the rationale for glutathione supplementation is grounded in biological plausibility and some preliminary scientific observations, robust clinical evidence for its effectiveness in treating or significantly improving autoimmune disorders is limited. As a result, L-glutathione is sometimes used as an adjunctive therapy to help manage oxidative stress in these conditions, but it is not considered a primary or proven treatment. More research is needed to fully validate its therapeutic role.

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