Evidence supporting the use of: L-taurine
For the health condition: Arrhythmia

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Synopsis

Source of validity: Scientific
Rating (out of 5): 2

L-taurine, a sulfur-containing amino acid, has some scientific basis for its use in supporting or treating arrhythmias, though the evidence is limited and not yet robust enough for strong clinical recommendations. Taurine is naturally present in high concentrations in the heart and has several physiological roles, including osmoregulation, membrane stabilization, and modulation of calcium signaling—an important factor in cardiac muscle contraction and electrophysiology.

Some animal studies and small human trials have suggested that taurine supplementation may help stabilize cardiac electrical activity and reduce the risk or severity of certain arrhythmias, particularly in people with underlying deficiencies or specific electrolytic imbalances. For example, a few clinical studies from the late 20th and early 21st century reported reduced frequency of premature ventricular contractions (PVCs) in patients given taurine. The proposed mechanisms include modulation of intracellular calcium and potassium handling, antioxidant effects, and inhibition of abnormal automaticity in cardiac cells.

However, most of the evidence is derived from small-scale or open-label studies rather than large, randomized controlled trials. Major cardiology guidelines do not currently endorse taurine as a standard therapy for arrhythmias. Nevertheless, taurine supplementation is generally considered safe at moderate doses, with minimal adverse effects reported.

In summary, the use of L-taurine for arrhythmia is supported by some scientific studies and plausible mechanistic rationale, but the overall quality of evidence is modest, warranting a rating of 2 out of 5.

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